Rheumatoid Arthritis - Natural Homeopathy Treatment
Posted by Aura Homeopathy on November 17th, 2019
Rheumatoid arthritis is a chronic inflammatory disease of the entire connective tissue (the tissue that provides the shape and support of different organs) that primarily affects the synovial membranes that line the capsules surrounding the joints.
His cause is unknown. It is part of what are called autoimmune diseases, that is, diseases in which the body's defense system is disrupted and produces antibodies against its own tissues that it considers to be foreign bodies (this situation can be compared to the phenomenon of rejection during transplants). On the other hand, there seems to be a genetic predisposition
SIGNS AND SYMPTOMS
The early phase includes joint pain (polyarthralgia), which is red, swollen, deformed and stiff, especially in the morning, with improvement during the day.
Joint destruction leads to significant deformities (hands and feet). The synovium, consisting of a single layer of cells, proliferates, constituting six to ten layers. It is the pannus that destroys the environment. The most commonly affected joints are those of the hands, wrists and feet. There are more rarely nodules (small hard balls) under the skin, specific to the disease.
The criteria for the diagnosis are those of the American College (7 criteria required).
1. Articular and periarticular morning stiffness for more than one hour.
2. Swelling of at least three joint groups.
3. Arthritis of the joints of the hands.
4. Symmetrical arrangement of arthritis.
5. Presence of rheumatoid nodules.
6. Presence of rheumatoid factor: reaction of the positive latex with a rate at least equal to 1/80, positive Waaler-Rose reaction with a rate at least equal to 1/32.
7. Characteristic radiological images on the hands and wrists.
At least 4 of these 7 criteria are required.
Criteria 1, 2, 3 and 4 must have been present for at least six weeks.
Unfortunately, the diagnostic criteria for ACR are often only fulfilled at a fairly advanced stage of RA, whereas lesions are already important.
This is why some authors have proposed to rely on:
• More clinical criteria flexible:
- Arthritis affecting three joints and no longer three groups.
- Bilateral pains at the pressure of phalangeal metatarsal.
• The detection of certain antibodies often present in the early stages of RA:
citrullinated anti-peptides, detected 75 times out of 100, and very specific.
• MRI of the hands and feet that visualizes bone and cartilage erosions that go unnoticed on conventional X-rays.
Evolution and prognosis
The evolution is towards an extension by successive pushes with handicapping formations of the joints of the feet and the hands. After several years of evolution, the connective tissue of other organs can be reached, especially in the lung (pleurisy), heart (pericarditis), small arteries (circulation disorders resulting in tingling, numbness ...).
RA has three main aspects:
- Progressive forms without net remissions (70%);
- Intermittent forms with relapses and remissions (25%);
- Malignant forms with severe rheumatism and extra-articular manifestations, mainly vascular involvement, subcutaneous nodules, pleurisy, pericarditis, Guerot-Siögren syndrome (5%).
The prognosis is always very reserved on the articular plane, although there are moderate forms and severe forms. Bone and joint destruction often result in significant deformities, especially in the hands and feet.
PR reduces life expectancy by an average of seven years for men and three years for women.
Certain mortality factors are increased in frequency, either because of RA, or because of the drugs administered to treat it:
- Cardiac or cerebrovascular accidents.
Impairment - Gut Diseases
PR: autoimmune disease
The mechanism of RA remains mysterious. It is generally considered an autoimmune disorder. Only two points are certain:
- The initial phenomenon is the immune response that precedes the inflammatory response;
- In this immune reaction, T cells play a major role.
T-lymphocytes are more often responsible for the autoimmune process than autoantibodies. They infiltrate the target organ and exert a pathogenic effect by cytotoxicity, release of cytokines and activation of macrophages.
Autoimmune diseases are always polyfactorial. Their development requires the union of several factors: genetic. immunological, environmental, hormonal and psychological.
Improvement of the disease by all the methods that T-cell action: immunosuppressive or T-depletion at the level of the thoracic duct.
During autoimmune diseases. HLA-DR molecules appear in large numbers on cells that serve as targets for the autoimmune reaction. This aberrant expression was found in RA where HLA-DR molecules are expressed on synovial cells (synoviocytes) and cartilage (chondrocytes).
Causes of RA
RA is a polyfactorial disease.
First and foremost environmental factors
Role of modern food
The current mode of human nutrition seems to me to be the major cause of the hyperpermeability of the hail and the formation of dangerous food and bacterial waste that cross the "enlarged meshes" of the intestinal mucosa.
- The consumption of animal milks and their derivatives.
- The consumption of domesticated cereals.
- The cooking of most products.
- The extraction of oils by methods that can be criticized.
- pollution by additives and other substances.
- Irradiation of many products.
- The fairly frequent deficiencies in minerals and vitamins.
To exercise its function which is the digestion of food, the small intestine has:
- Enzymes responsible for splitting the proteins into amino acids
- Mucus responsible for protecting the enterocytes against the aggressor agents present in the intestinal lumen.
However, it is quite possible that the enzymes are adapted to ancestral nutrition and not to current nutrition.
Initial thrust and subsequent relapses of RA are often triggered by stress. Stress acts on neurons and leads to neuropeptide release, some of which influences immune responses. The stresses probably induce a release of interferon y, substance causing three effects which favor the outbreaks:
Role of interferon
Another factor that can reduce intestinal leakiness is interferon-y. This mediator can bind to receptors at the basal end of the enterocytes (Adams et al, 1993), which leads to a disjunction of the links between the enterocytes. This results in an excessive passage of macromolecules between the enterocytes, without these being necrotic or damaged.
Gene of susceptibility related to the female sex
Since RA is three or four times more common in women than in men. The role of favoring medium-dose estrogens can be suspected (BRENNAN and SILMAN 1995). Indeed, it is known that RA is:
- rare before puberty and after menopause (low doses of estrogen).
- Is common in women during periods of genital activity (average doses of estrogen).
- Often knows a remission during a pregnancy, (very high doses of estrogen).
- Is rather improved by the pill (artificial estrogen inhibiting the production of natural estrogens).
Role of bacteria
Several bacteria have been accused of playing a causal role in RA: mycobacteria, Escherichia coli, Clostridium perfringens, streptococci, anaerobes. But the most suspicious is in my opinion Proteus mirabilis.
- The titre of the anti-Proteus mirabilis antibodies is significantly higher in RA than in inflammatory rheumatism tracts or in normal controls.
- The titre of antibodies increases during PR surges and decreases during remissions.
Role of modern food
Modern food could work through two related mechanisms.
- Modification of the bacterial flora of the hail with proliferation of a dangerous bacterium, causative agent of RA.
- Aggression of the small bowel mucosa resulting in lesions of the enterocytes and excessive permeability of the intestinal wall.
Although harmful products vary from one individual to another, cereals, animal milks and cooked foods are very often involved.
In favorable cases, the private diet improves the symptomatology which is awakened by the introduction of the dangerous food. The removal of the latter causes a remission of the PR.
The influence of diet on RA is demonstrated by two arguments. - Fasting often improves the PR:
The exclusion of certain foods often improves the RA: some patients spontaneously practice a restriction diet, especially milk, having noted that the consumption of milk exacerbated their symptomatology.
It is a common disease affecting almost 1% of the population, with a clear predominance of women (4 women for one man). The age of onset is on average 45 years old.
Blood tests show signs of inflammation (increased sedimentation rate and C-reactive protein). A marker of the disease, called rheumatoid factor, is evidenced by the latex test and the Waaler-Rose reaction. The radiographs are initially normal and then show the damage of the joints.
Cortisone was the first effective remedy in crisis, but taken too long it decalcifies bones, sclerose a lot of tissues, bleeds, causes stomach ulcers, diabetes and hypertension, makes the nervous system swell and excites , until the delirious puff. In fact, it relieves, but aggravates the disease.
Nonsteroidal anti-inflammatory drugs are also aggressive for the stomach and liver (sometimes toxic hepatitis).
Methotrexate (chemotherapy used for ovarian cancers), gold salts (past of fashion), hydrochloroquine (harmful effects on the eyes, muscles, heart, liver and skin) are also used. central nervous system), sulfazalazine (quite well tolerated, also giving good results in ulcerative colitis) and finally, very fashionable, immunosuppressors when the origin seems to be autoimmune (with significant side effects for results often disappointing).
Best Homeopathy Medicine For Rheumatoid Arthritis
-Bryonia alba, rhus toxicodendron, ferrum phosphoricum, 4 granules of every three to six times a day in jacked skins three times a day thereafter.
- Bryonia alba, exudation of the serous (formation of synovial or periarticular fluid), with dryness of the mucous membranes (dry mouth and thirst), sharp throbbing pains, aggravated by the slightest movement, improved by the rest, the cold applications and the pressure strong ; swollen, red, hot articulate, taut and shiny skin.
- Rhus toxicodendron, chronic involvement of muscles, tendons and periarticular serous bursae, little swelling, stiff, stiff and painful joints at rest, need to gently and often move the joint, worsening at rest (in bed or sitting ) and by the damp cold, improved by slow movements and by heat.
- Ferrum phosphoricum, slightly red and slightly swollen joints, slight pain aggravated by the movement, a characteristic being the frequent change of symptoms (one day is the knee, two days after it is the shoulder, once there is fever, the next day it's over). Subject tired and weakened.
- Apis mellifica, drug of any rapid onset swelling, pale, does not prevent the joint from bending, but pain type burn on the surface, improved by the cold.
- Belladonna, signs as for infection (extreme redness, radiant heat, pounding pain, fever and thirst).
- Ruta graveolens, pains (sensation of breakage) of the spine and limbs obliging to move constantly in the bed, improvement by the movement and the hot applications, aggravation by the wet and cold weather, by the lying position, great tired. Specific action on tendons, muscles and periosteum.
It is advisable to get as close as possible to the ancestral nutritional mode, without imposing too drastic restrictions that would put off the patient. The diet includes six essential guidelines:
-Remove all cereals, except rice and buckwheat
-Remove all animal milks and their derivatives
-Enjoy as much raw or cooked food at a temperature below 110 degrees.
-Choose if possible "organic" foods close to the original products. -Consume
pressed oils -Take magnesium salts, trace elements, vitamins at physiological doses and lactic ferments.
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The results obtained are 80%. This plan spans one year.
Mode of action of the hypo-toxic diet
It acts probably in three ways:
1) Modification of the intestinal flora with eradication or at least rarefaction of a dangerous bacterium.
2) Correction of the intestinal function thanks to a food to which enzymes and mucus are adapted. It follows:
- Better digestion of proteins leaving only a few peptides persist.
- A restoration of the tightness of the hail further reducing the amount of peptides crossing the intestinal barrier.
3) Restoration of the oral tolerance, because while in an abolished intestine, the bacterial antigens pass between the enterocytes to gain the general circulation, in a normal intestine, these antigens are captured by the cells M. In the first case, the antigen triggers an immunity response, in the second case a tolerance response.